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The most-cited finding is that adding the peptide to telomerase-negative human fetal fibroblast cultures induced expression of the telomerase catalytic subunit (hTERT), increased telomerase activity and lengthened telomeres, with treated cells reportedly exceeding the usual replicative limit

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Further, pre-treatment with selenite significantly prevented the radiation-induced mutation of the hprt locus in AA8 cells

Docosahexaenoic acid suppresses oxidative stress-induced autophagy and cell death via the AMPK-dependent signaling pathway in immortalized fischer rat schwann cells 1