Thus, using complementary pharmacological and chemogenetic approaches, we provide evidence that the modulation of GLP-1 signalling in the OB contributes to the maintenance of energy homeostasis, and that augmenting OB GLP-1 signalling can improve metabolic parameters deregulated in the context of obesity
Second, most available studies have relatively short follow-up (82, 84), making it difficult to distinguish transient state-dependent fluctuation from sustained sensory change
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This finding was strengthened by another study that reported partially reduced activity, measured by reduction of cell viability and markers of oxidative stress, when EGCG was co-treated with superoxide dismutase and catalase [76]
