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Cysteine availability is typically the rate-limiting factor for synthesis, which is why N-acetyl cysteine (NAC), a cysteine donor, is one of the most effective strategies for raising glutathione levels [2][9]

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The exact influence of circulating Th17 cells on depression remains unclear, but since Th17 cells can infiltrate the brain parenchyma without requiring VLA4 signals, it can be hypothesized that Th17 cells may reach the brain and synthesize IL-17 (68)