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Potency: Roughly 10 times stronger at receptor binding compared to native IGF-1

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In biochemical and preclinical models, the peptide appears to regulate endothelial activity, cell migration, and cytoskeletal organization through interactions with kinase signaling cascades and nitric oxide related mechanisms

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Indicatively, there is activation of the VEGFR2-Akt-eNOS signaling pathway without the need for other known ligands or shear stress and evidenced control of vasomotor tone by the activation of the Src-Caveolin-1-eNOS pathway