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Although an expected reduction in vasopressor requirement was observed with exogenous Ang II administration in the ATHOS-3 trial [28], some patients did not have this vasoconstrictive response, leading to the hypothesis that peptidases, such as DPP3, could be involved in the lack of response to exogenous Ang II [12, 29]

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(our blog post about BPC-157 specifically can be found here)

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