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Since genetic manipulations (e.g., knockdown, knockout, overexpression, etc.) are not possible in RBCs, pharmacological modulation of surface and intracellular proteins by specific antibodies, inhibitors, and agonists remains the only currently available approach

Its formation results in activation of initiator caspase-8 with further downstream recruitment of executioner caspase-3 by limited proteolysis [73, 75]

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